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	<title>Comments on: Individualized Intensity Zones - Part 2 - Respiratory System</title>
	<link>http://www.teamrunningfree.com/blog/2010/02/07/individualized-intensity-zones-part-2-respiratory-system/</link>
	<description>Running, Triathlon, Adeventure</description>
	<pubDate>Thu, 09 Feb 2012 04:00:21 +0000</pubDate>
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		<title>By: Arthur K</title>
		<link>http://www.teamrunningfree.com/blog/2010/02/07/individualized-intensity-zones-part-2-respiratory-system/#comment-16757</link>
		<author>Arthur K</author>
		<pubDate>Wed, 17 Mar 2010 00:44:57 +0000</pubDate>
		<guid>http://www.teamrunningfree.com/blog/2010/02/07/individualized-intensity-zones-part-2-respiratory-system/#comment-16757</guid>
		<description>Hey Sheryl,

Sorry, I meant the trachaea. 

Anyway, here is further scientific backing about the idea of a respiratory system limitation that could potentially cause the "central governor" (Noakes) to recruit less peripheral muscles in the legs to protect the vital organs and systems.

http://jp.physoc.org/content/537/1/2.full.pdf

Journal of Physiology

Teleologically, this reflex may have as its
fundamental goal the protection of oxygen
delivery to the respiratory muscles, thus
ensuring the ability to maintain pulmonary
ventilation, proper regulation of arterial
blood gases and pH and overall organismic
homeostasis. Presumably, as the ‘vital organ’
responsible for supporting pulmonary function,
perfusion of the respiratory muscles,
particularly during physiological states in
which there is competition for cardiac output
such as heavy submaximal and maximal
exercise, has priority over the locomotor
muscles. This subservience of active limb blood
flow may be similar to that previously
established for the arterial baroreflex during
large-muscle dynamic exercise (Rowell, 1997).
Specifically, under conditions in which widespread
vasodilatation has occurred, thus
threatening the maintenance of systemic
vascular resistance and arterial blood pressure,
arterial baroreflex deactivation (unloading) will
produce a strong reflex sympathetic
vasoconstriction targeted, at least in part, at
the active limbs. This vasoconstrictor drive can
be sufficiently strong as to produce vasoconstriction
in working locomotor muscles, thus
ensuring the maintenance of arterial perfusion
pressure.</description>
		<content:encoded><![CDATA[<p>Hey Sheryl,</p>
<p>Sorry, I meant the trachaea. </p>
<p>Anyway, here is further scientific backing about the idea of a respiratory system limitation that could potentially cause the &#8220;central governor&#8221; (Noakes) to recruit less peripheral muscles in the legs to protect the vital organs and systems.</p>
<p><a href="http://jp.physoc.org/content/537/1/2.full.pdf" rel="nofollow">http://jp.physoc.org/content/537/1/2.full.pdf</a></p>
<p>Journal of Physiology</p>
<p>Teleologically, this reflex may have as its<br />
fundamental goal the protection of oxygen<br />
delivery to the respiratory muscles, thus<br />
ensuring the ability to maintain pulmonary<br />
ventilation, proper regulation of arterial<br />
blood gases and pH and overall organismic<br />
homeostasis. Presumably, as the ‘vital organ’<br />
responsible for supporting pulmonary function,<br />
perfusion of the respiratory muscles,<br />
particularly during physiological states in<br />
which there is competition for cardiac output<br />
such as heavy submaximal and maximal<br />
exercise, has priority over the locomotor<br />
muscles. This subservience of active limb blood<br />
flow may be similar to that previously<br />
established for the arterial baroreflex during<br />
large-muscle dynamic exercise (Rowell, 1997).<br />
Specifically, under conditions in which widespread<br />
vasodilatation has occurred, thus<br />
threatening the maintenance of systemic<br />
vascular resistance and arterial blood pressure,<br />
arterial baroreflex deactivation (unloading) will<br />
produce a strong reflex sympathetic<br />
vasoconstriction targeted, at least in part, at<br />
the active limbs. This vasoconstrictor drive can<br />
be sufficiently strong as to produce vasoconstriction<br />
in working locomotor muscles, thus<br />
ensuring the maintenance of arterial perfusion<br />
pressure.</p>
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		<title>By: Sheryl Rubinoff</title>
		<link>http://www.teamrunningfree.com/blog/2010/02/07/individualized-intensity-zones-part-2-respiratory-system/#comment-16748</link>
		<author>Sheryl Rubinoff</author>
		<pubDate>Tue, 16 Mar 2010 13:12:51 +0000</pubDate>
		<guid>http://www.teamrunningfree.com/blog/2010/02/07/individualized-intensity-zones-part-2-respiratory-system/#comment-16748</guid>
		<description>Hello Arthur K. Your article was very interesting, however there was one incorrect fact you mentioned in your "Could it be my respiratory system is..". You mentioned about constriction of the esophagus. I'm sure this was a slip-up. The esophagus is a tube leading down into your stomach, whereas the trachea and bronchial (mainstem bronchus and its branches as it goes down into the lungs from bronchioles to the terminal respiratory unit or alveoli where gas exchange takes place). Normally the bronchioles either get restricted (constriction) making it difficult to breathe in as the tube is narrowed it lets in less oxygen, or there is accumulation of secretions which also acts as restriction, or both--as we see in the allergic patient. 
I am a retired respiratory therapist, and hope this knowledge was helpful.</description>
		<content:encoded><![CDATA[<p>Hello Arthur K. Your article was very interesting, however there was one incorrect fact you mentioned in your &#8220;Could it be my respiratory system is..&#8221;. You mentioned about constriction of the esophagus. I&#8217;m sure this was a slip-up. The esophagus is a tube leading down into your stomach, whereas the trachea and bronchial (mainstem bronchus and its branches as it goes down into the lungs from bronchioles to the terminal respiratory unit or alveoli where gas exchange takes place). Normally the bronchioles either get restricted (constriction) making it difficult to breathe in as the tube is narrowed it lets in less oxygen, or there is accumulation of secretions which also acts as restriction, or both&#8211;as we see in the allergic patient.<br />
I am a retired respiratory therapist, and hope this knowledge was helpful.</p>
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